Anti-NKG2A + C+ E antibody [20d5] (FITC) (ab24866)

Overview

  • Product nameAnti-NKG2A + C+ E antibody [20d5] (FITC)
    See all NKG2A + C+ E primary antibodies
  • Description
    Rat monoclonal [20d5] to NKG2A + C+ E (FITC)
  • ConjugationFITC. Ex: 493nm, Em: 528nm
  • Tested applicationsSuitable for: Flow Cytmore details
  • Species reactivity
    Reacts with: Mouse
  • Immunogen

    Tissue/ cell preparation: CHO cells transfected with murine NKG2.

Properties

Applications

Our Abpromise guarantee covers the use of ab24866 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

Application Abreviews Notes
Flow Cyt
  • Application notesFlow Cyt: Use 1µg for 106 cells.

    Not yet tested in other applications.
    Optimal dilutions/concentrations should be determined by the end user.
  • Target

    • RelevanceNKG2A, NKG2C and NKG2E, are isoforms of the NKG2 lectin-like family. In mice, NKG2 subunits associate with CD94 to form heterodimers at the surface of natural killer (NK) cells.The CD94/NKG2 heterodimer is the receptor for a non-classical MHC class I ligand, which is Qa 1 in the mouse. NKG2 is expressed on NK cells, lymphokine-activated killer (LAK) T cells, and some CD8+ memory T cells.
    • Cellular localizationType II membrane protein.
    • Database links
    • Alternative names
      • KLRC1 antibody
      • KLRC2 antibody
      • KLRC3 antibody
      • NK cell receptor A antibody
      • NK cell receptor C antibody
      • NK cell receptor E antibody
      • NKG2 A activating NK receptor antibody
      • NKG2 A type II integral membrane protein antibody
      • NKG2 C activating NK receptor antibody
      • NKG2 C type II integral membrane protein antibody
      • NKG2 E activating NK receptor antibody
      • NKG2 E type II integral membrane protein antibody
      see all

    References for Anti-NKG2A + C+ E antibody [20d5] (FITC) (ab24866)

    ab24866 has not yet been referenced specifically in any publications.

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