Overview

  • Product nameAnti-RECQ1 antibody
    See all RECQ1 primary antibodies
  • Description
    Mouse polyclonal to RECQ1
  • Tested applicationsSuitable for: WBmore details
  • Species reactivity
    Reacts with: Human
  • Immunogen

    Full length protein, corresponding to amino acids 1 - 694 of Human RECQ1 (NP_002898.2)

  • Positive control
    • HeLa cells nuclear lysate

Properties

Associated products

Applications

Our Abpromise guarantee covers the use of ab89817 in the following tested applications.

The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.

Application Abreviews Notes
WB Use a concentration of 1 µg/ml. Predicted molecular weight: 73 kDa.

Target

  • RelevanceThe protein encoded by this gene is a member of the RecQ DNA helicase family. DNA helicases are enzymes involved in various types of DNA repair, including mismatch repair, nucleotide excision repair and direct repair. Some members of this family are associated with genetic disorders with predisposition to malignancy and chromosomal instability.
  • Cellular localizationNuclear
  • Database links
  • Alternative names
    • ATP dependent DNA helicase Q1 antibody
    • DNA helicase Q1 like antibody
    • DNA helicase RecQ like type 1 antibody
    • RECQ L1 antibody
    • RecQ protein like antibody
    • RecQ protein like 1 antibody
    • RECQL antibody
    • RECQL1 antibody
    see all

Anti-RECQ1 antibody images

  • Anti-RECQ1 antibody (ab89817) at 1 µg/ml + HeLa cells nuclear extracts at 50 µg

    Predicted band size : 73 kDa
    Observed band size : 73 kDa
  • All lanes : Anti-RECQ1 antibody (ab89817) at 1 µg/ml

    Lane 1 : RECQ1 transfected 293T cell lysate
    Lane 2 : Non-transfected lysate

    Lysates/proteins at 25 µg per lane.


    Predicted band size : 73 kDa
  • ab89817 at 10µg/ml staining RECQ1 in PFA-fixed, permeabilized HeLa cells by immunofluorescence.

References for Anti-RECQ1 antibody (ab89817)

ab89817 has not yet been referenced specifically in any publications.

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