Key features and details
- Alexa Fluor® 647 Mouse monoclonal [24F.10C12] to PD-L2
- Suitable for: Flow Cyt
- Reacts with: Human
- Conjugation: Alexa Fluor® 647. Ex: 652nm, Em: 668nm
- Isotype: IgG2a
Product nameAlexa Fluor® 647 Anti-PD-L2 antibody [24F.10C12]
See all PD-L2 primary antibodies
DescriptionAlexa Fluor® 647 Mouse monoclonal [24F.10C12] to PD-L2
ConjugationAlexa Fluor® 647. Ex: 652nm, Em: 668nm
Tested applicationsSuitable for: Flow Cytmore details
Species reactivityReacts with: Human
Full length protein corresponding to Human PD-L2.
- Flow Cyt: Human monocyte-derived dendritic cells.
Storage instructionsShipped at 4°C. Store at +4°C. Store In the Dark.
Storage bufferpH: 7.4
Preservative: 0.0975% Sodium azide
Concentration information loading...
Purification notesThe purified antibody is conjugated with Alexa Fluor® 647 under optimum conditions. The conjugate is purified by size-exclusion chromatography and adjusted for direct use. No reconstitution is necessary.
Our Abpromise guarantee covers the use of ab243086 in the following tested applications.
|Flow Cyt||Use 4µl for 106 cells.
OR 100 µl of whole blood.
FunctionInvolved in the costimulatory signal, essential for T-cell proliferation and IFNG production in a PDCD1-independent manner. Interaction with PDCD1 inhibits T-cell proliferation by blocking cell cycle progression and cytokine production.
Tissue specificityHighly expressed in heart, placenta, pancreas, lung and liver and weakly expressed in spleen, lymph nodes and thymus.
Sequence similaritiesBelongs to the immunoglobulin superfamily. BTN/MOG family.
Contains 1 Ig-like C2-type (immunoglobulin-like) domain.
Contains 1 Ig-like V-type (immunoglobulin-like) domain.
Cellular localizationSecreted; Cell membrane and Endomembrane system.
- Information by UniProt
- B7 dendritic cell molecule antibody
- B7-DC antibody
- B7DC antibody
To our knowledge, customised protocols are not required for this product. Please try the standard protocols listed below and let us know how you get on.
ab243086 has not yet been referenced specifically in any publications.