Product nameAnti-CCR7 antibody [4B12]
See all CCR7 primary antibodies
DescriptionRat monoclonal [4B12] to CCR7
Tested applicationsSuitable for: Flow Cyt, IPmore details
Species reactivityReacts with: Mouse
Tissue, cells or virus corresponding to Mouse CCR7. (Murine CD197-transfected RBL-2H3 cells).
Storage instructionsShipped at 4°C. Store at +4°C short term (1-2 weeks). Upon delivery aliquot. Store at -20°C long term. Avoid freeze / thaw cycle.
Storage bufferpH: 7.40
Preservative: 0.097% Sodium azide
Constituent: 99% PBS
Concentration information loading...
PurityProtein G purified
Purification notesab188426 was purified from cell culture supernatant. Purity is > 95% (by SDS-PAGE).
- Anti-CCR7 antibody [4B12] - Low endotoxin, Azide free (ab185745)
- Anti-CCR7 antibody [4B12] (Phycoerythrin) (ab230665)
- Anti-CCR7 antibody [4B12] - BSA and Azide free (ab269572)
- Anti-CCR7 antibody [4B12] (ab52602)
- Anti-CCR7 antibody [4B12] (APC) (ab93518)
- Anti-CCR7 antibody [4B12] (Biotin) (ab93519)
- Anti-CCR7 antibody [4B12] (Phycoerythrin) (ab95669)
Our Abpromise guarantee covers the use of ab188426 in the following tested applications.
The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.
|Flow Cyt||Use at an assay dependent concentration.
ab18450 - Rat monoclonal IgG2a, is suitable for use as an isotype control with this antibody.
|IP||Use at an assay dependent concentration.|
FunctionReceptor for the MIP-3-beta chemokine. Probable mediator of EBV effects on B-lymphocytes or of normal lymphocyte functions.
Tissue specificityExpressed in various lymphoid tissues and activated B- and T-lymphocytes, strongly up-regulated in B-cells infected with Epstein-Barr virus and T-cells infected with herpesvirus 6 or 7.
Sequence similaritiesBelongs to the G-protein coupled receptor 1 family.
Cellular localizationCell membrane.
- Information by UniProt
- BLR 2 antibody
- BLR2 antibody
- C C chemokine receptor type 7 antibody
ab188426 has not yet been referenced specifically in any publications.