(R,S)-Sulforaphane, antagonist of Ah receptor

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(4 Publications)

MW 177.3 Da, Purity >98%. Potent, non-competitive antagonist of the aryl hydrocarbon (Ah) receptor (IC₅₀ = 1 μM). Isothiocyanate. Induces epoxide hydrolase. Anticarcinogenic. Active in vivo and in vitro.

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Azoreductase, Cytochrome b 5 reductase, DHQU, DIA 4, DT-diaphorase, DTD, Diaphorase (NADH/NADPH), Diaphorase (NADH/NADPH) (cytochrome b 5 reductase), Diaphorase 4, Dioxin inducible 1, HEP-NOS, Hepatocyte NOS, Inducible NO synthase, Inducible NOS, Inducible nitric oxide synthase, MAC NOS, Macrophage NOS, Menadione reductase, NAD(P)H dehydrogenase [quinone] 1, NAD(P)H dehydrogenase quinone 1, NAD(P)H menadione oxidoreductase 1 dioxin inducible, NAD(P)H quinone dehydrogenase 1, NAD(P)H: menadione oxidoreductase 1 dioxin inducible 1, NAD(P)H:Quinone acceptor oxidoreductase type 1, NAD(P)H:menadione oxidoreductase 1, NAD(P)H:quinone oxidoreductase 1, NAD(P)H:quinone oxireductase, NF-E2-related factor 2, NF2L2_HUMAN, NMOR 1, NMOR I, NOS, NOS type II, NOS2A, NOS2A, Inducible, Hepatocyte, NOS2_HUMAN, NQO1_HUMAN, NRF2, Nfe2l2, Nitric oxide synthase, Nitric oxide synthase 2 inducible, Nitric oxide synthase 2 inducible macrophage, Nitric oxide synthase inducible, Nos II, Nuclear factor, Nuclear factor (erythroid derived 2) like 2, Nuclear factor erythroid 2-related factor 2, Nuclear factor erythroid derived 2 like 2, Peptidyl-cysteine S-nitrosylase NOS2, Phylloquinone reductase, QR 1, Quinone reductase 1, erythroid derived 2, iNOS, inducible, like 2, nitric oxide synthase 2A (inducible, hepatocytes), nitric oxide synthase, macrophage, nuclear factor erythroid 2 like 2