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ARL2BP

Function

Together with ARL2, plays a role in the nuclear translocation, retention and transcriptional activity of STAT3. May play a role as an effector of ARL2.

Involvement in disease

Retinitis pigmentosa 82 with or without situs inversus

RP82

An autosomal recessive disorder characterized by variable association of retinitis pigmentosa with situs inversus. Retinitis pigmentosa is characterized by retinal pigment deposits visible on fundus examination and primary loss of rod photoreceptor cells followed by secondary loss of cone photoreceptors. Patients typically have night vision blindness and loss of midperipheral visual field. As their condition progresses, they lose their far peripheral visual field and eventually central vision as well. Situs inversus is a congenital abnormality in which organs in the thorax and the abdomen are opposite to their normal positions due to lateral transposition.

None

The disease is caused by variants affecting the gene represented in this entry.

Sequence Similarities

Belongs to the ARL2BP family.

Tissue Specificity

Expressed in retina pigment epithelial cells (at protein level). Widely expressed.

Cellular localization

Alternative names

BART, BART1, ARL2BP, ADP-ribosylation factor-like protein 2-binding protein, ARF-like 2-binding protein, ARL2-binding protein, Binder of ARF2 protein 1

swissprot:Q9Y2Y0 omim:615407 entrezGene:23568