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CLCN3

Domain

Isoform 2 contains a C-terminal PDZ-binding motif mediating the interaction with GOPC.

Function

Isoform 1

Strongly outwardly rectifying, electrogenic H(+)/Cl(-)exchanger which mediates the exchange of chloride ions against protons (By similarity). The CLC channel family contains both chloride channels and proton-coupled anion transporters that exchange chloride or another anion for protons (PubMed:29845874). The presence of conserved gating glutamate residues is typical for family members that function as antiporters (PubMed:29845874).

Isoform 2

Strongly outwardly rectifying, electrogenic H(+)/Cl(-)exchanger which mediates the exchange of chloride ions against protons.

Involvement in disease

Neurodevelopmental disorder with hypotonia and brain abnormalities

NEDHYBA

An autosomal dominant disorder characterized by onset in infancy or early childhood, global developmental delay, hypotonia, impaired intellectual development, and poor or absent speech. Additional variable manifestations may be present, including feeding difficulties, seizures, behavioral abnormalities, and non-specific dysmorphic facial features. Brain imaging shows variable abnormalities, including corpus callosum and cerebellar defects, and decreased white matter volume.

None

The disease is caused by variants affecting the gene represented in this entry.

Neurodevelopmental disorder with seizures and brain abnormalities

NEDSBA

An autosomal recessive neurologic disorder characterized by global developmental delay and onset of seizures in the first months of life, and structural brain defects on brain imaging. Additional features may include pigmentary retinopathy with poor visual fixation and spasticity.

None

The disease is caused by variants affecting the gene represented in this entry.

Post-translational modifications

N-glycosylated.

Sequence Similarities

Belongs to the chloride channel (TC 2.A.49) family. ClC-3/CLCN3 subfamily.

Tissue Specificity

Expressed primarily in tissues derived from neuroectoderm. Within the brain, its expression is particularly evident in the hippocampus, olfactory cortex, and olfactory bulb. Highly expressed in aortic and coronary vascular smooth muscle cells, and aortic endothelial cells. Also expressed in tracheal and alveolar epithelial cells, and intima and media of the pulmonary vessels. Expressed in bronchus and colon (at protein level).

Cellular localization

Alternative names

H(+)/Cl(-) exchange transporter 3, Chloride channel protein 3, Chloride transporter ClC-3, ClC-3, CLCN3

swissprot:P51790