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DAGLA

Function

Serine hydrolase that hydrolyzes arachidonic acid-esterified diacylglycerols (DAGs) to produce the principal endocannabinoid, 2-arachidonoylglycerol (2-AG) (PubMed:14610053, PubMed:23502535, PubMed:26668358). Preferentially hydrolyzes sn-1 fatty acids from diacylglycerols (DAG) that contain arachidonic acid (AA) esterified at the sn-2 position to biosynthesize 2-AG (PubMed:14610053, PubMed:23502535, PubMed:26668358). Has negligible activity against other lipids including monoacylglycerols and phospholipids (PubMed:14610053). Plays a key role in regulating 2-AG signaling in the central nervous system (CNS). Regulates 2-AG involved in retrograde suppression at central synapses. Supports axonal growth during development and adult neurogenesis. Plays a role for eCB signaling in the physiological regulation of anxiety and depressive behaviors. Regulates also neuroinflammatory responses in the brain, in particular, LPS-induced microglial activation (By similarity).

Involvement in disease

Spinocerebellar ataxia 20

SCA20

Spinocerebellar ataxia is a clinically and genetically heterogeneous group of cerebellar disorders. Patients show progressive incoordination of gait and often poor coordination of hands, speech and eye movements, due to degeneration of the cerebellum with variable involvement of the brainstem and spinal cord. SCA20 is an autosomal dominant, adult-onset form characterized by dysarthria due to spasmodic dysphonia followed by slowly progressive ataxia.

None

The disease may be caused by variants affecting the gene represented in this entry. A copy number variation consisting of a 260-kb duplication at chromosome 11q12.2-12.3 is responsible for SCA20. The critical gene within the duplicated segment may be DAGLA.

Neuroocular syndrome 2, paroxysmal type

NOC2

A form of neuroocular syndrome, a group of disorders characterized by developmental delay, impaired intellectual development and ocular anomalies as primary findings. NOC2 is an autosomal dominant form characterized by eye deviation or nystagmus with abnormal head posturing apparent from birth or early infancy. Affected individuals also have hypotonia, mild developmental delay, dysarthria, and gait ataxia. Most patients have mildly impaired intellectual development.

None

The disease is caused by variants affecting the gene represented in this entry.

Post-translational modifications

Phosphorylated at Ser-782 and Ser-808 by CAMK2A; phosphorylation by CAMK2A inhibits diacylglycerol lipase activity.

Sequence similarities

Belongs to the AB hydrolase superfamily. Lipase family.

Tissue specificity

Highly expressed in brain and pancreas.

Cellular localization

  • Cell membrane
  • Multi-pass membrane protein
  • Postsynaptic density membrane
  • Multi-pass membrane protein
  • Early endosome membrane
  • Multi-pass membrane protein
  • Cell projection
  • Dendritic spine membrane
  • Multi-pass membrane protein
  • Cycles between the cell surface and an intracellular endosomal compartment. Internalized by early endosomes via a clathrin-independent pathway before transport back to the postsynaptic membrane surface in a PKC-dependent manner.

Alternative names

  • C11orf11
  • KIAA0659
  • NSDDR
  • DAGLA
  • C11orf11
  • KIAA0659
  • NSDDR
  • Diacylglycerol lipase-alpha
  • DAGL-alpha
  • DGL-alpha
  • Neural stem cell-derived dendrite regulator
  • Sn1-specific diacylglycerol lipase alpha

Target type

Proteins

Molecular weight

114952Da

We found 1 product in 1 category

Primary Antibodies

Target

Application

Reactive species

Search our catalogue for 'DAGLA' (1)

Products