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Function

Structural component of gap junctions (PubMed:16849369, PubMed:17551008, PubMed:19340074, PubMed:19384972, PubMed:21094651, PubMed:26753910). Gap junctions are dodecameric channels that connect the cytoplasm of adjoining cells. They are formed by the docking of two hexameric hemichannels, one from each cell membrane (PubMed:17551008, PubMed:19340074, PubMed:21094651, PubMed:26753910). Small molecules and ions diffuse from one cell to a neighboring cell via the central pore (PubMed:16849369, PubMed:19384972, PubMed:21094651).

Involvement in disease

Deafness, autosomal recessive, 1A

DFNB1A

A form of non-syndromic sensorineural hearing loss. Sensorineural deafness results from damage to the neural receptors of the inner ear, the nerve pathways to the brain, or the area of the brain that receives sound information.

None

The disease is caused by variants affecting the gene represented in this entry.

Deafness, autosomal dominant, 3A

DFNA3A

A form of non-syndromic sensorineural hearing loss. Sensorineural deafness results from damage to the neural receptors of the inner ear, the nerve pathways to the brain, or the area of the brain that receives sound information.

None

The disease is caused by variants affecting the gene represented in this entry.

Vohwinkel syndrome

VOWNKL

An autosomal dominant disease characterized by hyperkeratosis, constriction on fingers and toes and congenital deafness.

None

The disease is caused by variants affecting the gene represented in this entry.

Keratoderma, palmoplantar, with deafness

PPKDFN

An autosomal dominant disorder characterized by the association of palmoplantar hyperkeratosis with progressive, bilateral, high-frequency, sensorineural deafness.

None

The disease is caused by variants affecting the gene represented in this entry.

Keratitis-ichthyosis-deafness syndrome, autosomal dominant

KIDAD

An autosomal dominant form of keratitis-ichthyosis-deafness syndrome, a disease characterized by the association of hyperkeratotic skin lesions with vascularizing keratitis and profound sensorineural hearing loss. Clinical features include deafness, ichthyosis, photophobia, absent or decreased eyebrows, sparse or absent scalp hair, decreased sweating and dysplastic finger and toenails.

None

The disease is caused by variants affecting the gene represented in this entry.

Bart-Pumphrey syndrome

BAPS

An autosomal dominant disorder characterized by sensorineural hearing loss, palmoplantar keratoderma, knuckle pads, and leukonychia, It shows considerable phenotypic variability.

None

The disease is caused by variants affecting the gene represented in this entry.

Ichthyosis hystrix-like with deafness syndrome

HID syndrome

An autosomal dominant keratinizing disorder characterized by sensorineural deafness and spiky hyperkeratosis affecting the entire skin. HID syndrome is considered to differ from the similar KID syndrome in the extent and time of occurrence of skin symptoms and the severity of the associated keratitis.

None

The disease is caused by variants affecting the gene represented in this entry.

Sequence similarities

Belongs to the connexin family. Beta-type (group I) subfamily.

Cellular localization

  • Cell membrane
  • Multi-pass membrane protein
  • Cell junction
  • Gap junction
  • Colocalizes with GJB4 at gap junction plaques in the cochlea.

Alternative names

Gap junction beta-2 protein, Connexin-26, Cx26, GJB2

Target type

Proteins

Primary research area

Neuroscience

Molecular weight

26215Da

We found 2 products in 2 categories

Primary Antibodies

Target

Application

Reactive species

Proteins & Peptides

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Species of origin

Search our catalogue for 'Gjb2' (2)

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