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MYBPC3

Function

Thick filament-associated protein located in the crossbridge region of vertebrate striated muscle a bands. In vitro it binds MHC, F-actin and native thin filaments, and modifies the activity of actin-activated myosin ATPase. It may modulate muscle contraction or may play a more structural role.

Involvement in disease

Cardiomyopathy, familial hypertrophic, 4

CMH4

A hereditary heart disorder characterized by ventricular hypertrophy, which is usually asymmetric and often involves the interventricular septum. The symptoms include dyspnea, syncope, collapse, palpitations, and chest pain. They can be readily provoked by exercise. The disorder has inter- and intrafamilial variability ranging from benign to malignant forms with high risk of cardiac failure and sudden cardiac death.

None

The disease is caused by variants affecting the gene represented in this entry.

Cardiomyopathy, dilated, 1MM

CMD1MM

A disorder characterized by ventricular dilation and impaired systolic function, resulting in congestive heart failure and arrhythmia. Patients are at risk of premature death.

None

The disease is caused by variants affecting the gene represented in this entry.

MYBPC3 mutations may be involved in restrictive cardiomyopathy (RCM), a rare non-ischemic myocardial disease. RCM is characterized by restrictive ventricular-filling physiology in the presence of normal or reduced diastolic and/or systolic volumes (of 1 or both ventricles), biatrial enlargement, and normal ventricular wall thickness.

Left ventricular non-compaction 10

LVNC10

A form of left ventricular non-compaction, a cardiomyopathy due to myocardial morphogenesis arrest and characterized by a hypertrophic left ventricle, a severely thickened 2-layered myocardium, numerous prominent trabeculations, deep intertrabecular recesses, and poor systolic function. Clinical manifestations are variable. Some affected individuals experience no symptoms at all, others develop heart failure. In some cases, left ventricular non-compaction is associated with other congenital heart anomalies. LVNC10 is an autosomal dominant condition.

None

The disease is caused by variants affecting the gene represented in this entry.

Post-translational modifications

Substrate for phosphorylation by PKA and PKC. Reversible phosphorylation appears to modulate contraction (By similarity).

Polyubiquitinated.

Sequence similarities

Belongs to the immunoglobulin superfamily. MyBP family.

Alternative names

  • Cardiac MyBP-C
  • MYBPC3

Target type

Proteins

Primary research area

Cardiovascular

Molecular weight

140762Da

We found 11 products in 3 categories

Primary Antibodies

Target

Application

Reactive species

Assay Kits

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Reactive species

Proteins & Peptides

Target

Species of origin

Nature