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SLCO1B3

Function

Mediates the Na(+)-independent uptake of organic anions such as 17-beta-glucuronosyl estradiol, taurocholate, triiodothyronine (T3), leukotriene C4, dehydroepiandrosterone sulfate (DHEAS), methotrexate and sulfobromophthalein (BSP). Involved in the clearance of bile acids and organic anions from the liver.

Involvement in disease

Hyperbilirubinemia, Rotor type

HBLRR

An autosomal recessive form of primary conjugated hyperbilirubinemia. Affected individuals develop mild jaundice not associated with hemolysis shortly after birth or in childhood. They have delayed plasma clearance of the unconjugated anionic dye bromsulphthalein and prominent urinary excretion of coproporphyrin I. Hepatic pigmentation is normal.

None

The disease is caused by variants affecting the gene represented in this entry.

Post-translational modifications

N-glycosylated.

Sequence similarities

Belongs to the organo anion transporter (TC 2.A.60) family.

Tissue specificity

Highly expressed in liver, in particular at the basolateral membrane of hepatocytes near the central vein. Not detected in other tissues. Highly expressed in some cancer cell lines derived from colon, pancreas, liver and gall bladder.

Cellular localization

  • Basolateral cell membrane
  • Multi-pass membrane protein

Alternative names

  • Solute carrier organic anion transporter family member 1B3
  • Liver-specific organic anion transporter 2
  • Organic anion transporter 8
  • Organic anion-transporting polypeptide 8
  • Solute carrier family 21 member 8
  • LST-2
  • OATP-8
  • SLCO1B3
  • LST2
  • OATP1B3
  • SLC21A8
  • OATP8

Target type

Proteins

Molecular weight

77403Da