Key features and details
- Rabbit Polyclonal to Kallikrein 4
- Suitable for: IHC-P
- Reacts with: Human
- Isotype: IgG
Product nameAnti-Kallikrein 4 antibody
See all Kallikrein 4 primary antibodies
DescriptionRabbit Polyclonal to Kallikrein 4
Specificityab197657 detects endogenous level of total Kallikrein 4 protein.
Tested applicationsSuitable for: IHC-Pmore details
Species reactivityReacts with: Human
Predicted to work with: Mouse
Fusion protein within Human Kallikrein 4 (internal sequence). The exact sequence is proprietary. Fusion protein corresponding to a region derived from internal residues of human Kallikrein 4. NCBI Accession No. BC069429. The protein fusion partner is GST.
Database link: Q9Y5K2
- Human cervical cancer and thyroid cancer tissues.
Storage instructionsShipped at 4°C. Store at +4°C short term (1-2 weeks). Upon delivery aliquot. Store at -20°C long term. Avoid freeze / thaw cycle.
Storage bufferpH: 7.4
Preservative: 0.05% Sodium azide
Constituents: 50% Glycerol (glycerin, glycerine), 49% PBS
Concentration information loading...
PurityImmunogen affinity purified
Our Abpromise guarantee covers the use of ab197657 in the following tested applications.
The application notes include recommended starting dilutions; optimal dilutions/concentrations should be determined by the end user.
|IHC-P||1/15 - 1/100. Perform heat mediated antigen retrieval with citrate buffer pH 6 before commencing with IHC staining protocol.|
Tissue specificityExpressed in prostate.
Involvement in diseaseDefects in KLK4 are the cause of amelogenesis imperfecta hypomaturation type 2A1 (AI2A1) [MIM:204700]. AI2A1 is an autosomal recessive defect of enamel formation. The disorder involves both primary and secondary dentitions. The teeth have a shiny agar jelly appearance and the enamel is softer than normal. Brown pigment is present in middle layers of enamel.
Sequence similaritiesBelongs to the peptidase S1 family. Kallikrein subfamily.
Contains 1 peptidase S1 domain.
- Information by UniProt
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ab197657 has not yet been referenced specifically in any publications.