Publishing research using ab84078? Please let us know so that we can cite the reference in this datasheet.

ab84078 has been referenced in 9 publications.

  • An H  et al. ALS-linked FUS mutations confer loss and gain of function in the nucleus by promoting excessive formation of dysfunctional paraspeckles. Acta Neuropathol Commun 7:7 (2019). PubMed: 30642400
  • Fujita K  et al. Targeting Tyro3 ameliorates a model of PGRN-mutant FTLD-TDP via tau-mediated synaptic pathology. Nat Commun 9:433 (2018). PubMed: 29382817
  • Luisier R  et al. Intron retention and nuclear loss of SFPQ are molecular hallmarks of ALS. Nat Commun 9:2010 (2018). PubMed: 29789581
  • West JA  et al. Structural, super-resolution microscopy analysis of paraspeckle nuclear body organization. J Cell Biol 214:817-30 (2016). PubMed: 27646274
  • Di Salvio M  et al. Pur-alpha functionally interacts with FUS carrying ALS-associated mutations. Cell Death Dis 6:e1943 (2015). PubMed: 26492376
  • Lenzi J  et al. ALS mutant FUS proteins are recruited into stress granules in induced pluripotent stem cell-derived motoneurons. Dis Model Mech 8:755-66 (2015). PubMed: 26035390
  • Kukharsky MS  et al. Calcium-responsive transactivator (CREST) protein shares a set of structural and functional traits with other proteins associated with amyotrophic lateral sclerosis. Mol Neurodegener 10:20 (2015). PubMed: 25888396
  • Shelkovnikova TA  et al. Compromised paraspeckle formation as a pathogenic factor in FUSopathies. Hum Mol Genet 23:2298-312 (2014). PubMed: 24334610
  • Wang WY  et al. Interaction of FUS and HDAC1 regulates DNA damage response and repair in neurons. Nat Neurosci 16:1383-91 (2013). PubMed: 24036913

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