Publishing research using ab33499? Please let us know so that we can cite the reference in this datasheet.

ab33499 has been referenced in 11 publications.

  • Normanno D  et al. Mutational phospho-mimicry reveals a regulatory role for the XRCC4 and XLF C-terminal tails in modulating DNA bridging during classical non-homologous end joining. Elife 6:N/A (2017). Human . PubMed: 28500754
  • Harada K  et al. Gimeracil enhances the antitumor effect of cisplatin in oral squamous cell carcinoma cellsin vitroandin vivo. Oncol Lett 14:3349-3356 (2017). PubMed: 28927087
  • Simon NE  et al. RNA-binding protein RBM14 regulates dissociation and association of non-homologous end joining proteins. Cell Cycle 16:1175-1180 (2017). PubMed: 28426349
  • Shamanna RA  et al. WRN regulates pathway choice between classical and alternative non-homologous end joining. Nat Commun 7:13785 (2016). PubMed: 27922005
  • Ochi T  et al. DNA repair. PAXX, a paralog of XRCC4 and XLF, interacts with Ku to promote DNA double-strand break repair. Science 347:185-8 (2015). PubMed: 25574025
  • Reid DA  et al. Organization and dynamics of the nonhomologous end-joining machinery during DNA double-strand break repair. Proc Natl Acad Sci U S A 112:E2575-84 (2015). PubMed: 25941401
  • Guo C  et al. XRCC4 deficiency in human subjects causes a marked neurological phenotype but no overt immunodeficiency. J Allergy Clin Immunol 136:1007-17 (2015). PubMed: 26255102
  • Terasawa M  et al. Canonical Non-Homologous End Joining in Mitosis Induces Genome Instability and Is Suppressed by M-phase-Specific Phosphorylation of XRCC4. PLoS Genet 10:e1004563 (2014). WB ; Human . PubMed: 25166505
  • Britton S  et al. A new method for high-resolution imaging of Ku foci to decipher mechanisms of DNA double-strand break repair. J Cell Biol 202:579-95 (2013). WB ; Mouse, Human . PubMed: 23897892
  • Roy S  et al. XRCC4's interaction with XLF is required for coding (but not signal) end joining. Nucleic Acids Res 40:1684-1694 (2012). WB ; Human . PubMed: 22228831
  • Slupianek A  et al. BCR/ABL stimulates WRN to promote survival and genomic instability. Cancer Res 71:842-51 (2011). WB ; Human . PubMed: 21123451

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